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In addition, ROS activate inflammasomes and promote pyroptotic cell death, further contributing to bone loss and progressive tissue destruction ( In peri-implant tissues, these effects may be amplified due to inherent structural and functional differences, contributing to increased susceptibility to ROS-mediated degeneration and potentially explaining the more aggressive and treatment-resistant nature of peri-implantitis ( Building on this, it is important to consider that much of the current understanding of redox dysregulation is derived from periodontitis models

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Rheumatic Disease Clinics of North America